Poxviruses

Name: Poxviruses
Brand: Springer-Verlag Berlin and Heidelberg GmbH & Co. KG
SKU: 9783642756078
Price: 106.17 EUR
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Autor: Richard W. Moyer, Peter C. Turner

Jazyk:

Väzba: Brožovaná

Vydavateľ: Springer-Verlag Berlin and Heidelberg GmbH & Co. KG

Dostupnosť: Skladom u dodávateľa v malom množstve

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The purpose of this volume is to highlight some current areas of poxvirus research which are likely...

Informácie o knihe

Autor

Richard W. Moyer, Peter C. Turner

Jazyk

Angličtina

Väzba

Kniha - Brožovaná

Vydalo

2011

Stránok

211

EAN

9783642756078

ISBN

3642756077

Enbook ID

07057910

Vydavateľ

Springer-Verlag Berlin and Heidelberg GmbH & Co. KG

Hmotnosť

359

Rozmery

155 x 235 x 13

Kompletný popis

The purpose of this volume is to highlight some current areas of poxvirus research which are likely to be particularly fruitful in the upcoming few years. The first chapter, by Drs. Condit and Niles, discusses poxvirus genetics. Work in this area has provided mutants, produced practical procedures to simplify the manipulation of viral genes, and generated information about the molecular architecture and organization of genes characteristic of pox viruses. One of the most intensively studied regions of the viral genome is the HindIII D region of vaccinia, in which a combination of classical and molecular genetic analysis of the region has been particularly revealing. Within this region are open reading frames, some of which are expressed early and others late, organized in a fashion which is now known to be typical of these viruses. Other studies, related to temperature sensitive, drug resistant, and drug dependent mutants, are also discussed. Each of the other reviews included in this volume summarizes areas of research which have depended heavily on the genetics of the system. The intracellular site of a poxvirus infection is mostly, if not exclusively, limited to the cytoplasm which dictates several interesting biological ramifications. For example, poxvirus transcription must occur in the cytoplasm, rather than in the nucleus. The virus copes with this situation by incorporating into the virion the enzymatic machinery necessary to initiate transcription from input virus.

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